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SQSTM1 and its MAP1LC3B-binding domain induce forced mitophagy to degrade mitochondrial carryover during mitochondrial replacement therapy

The presence of these toxic byproducts can cause damage to liver cells, increasing the risk of liver injury or failure

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Future research could explore the molecular mechanisms underlying the effects of specific amino acids on stress responses, the long-term effects of dietary amino acid manipulation, and the potential for genetic approaches to enhance amino acid utilization

In accordance with this, associations between chronological age and a higher ROS production rate of mitochondria in brain cells were demonstrated in rodents (Navarro and Boveris, 2004

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