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Moreover, these exosomes partially inhibit premature activation and depletion of primordial follicles by modulating the PI3K/AKT/mTOR signaling pathway, which is typically hyperactivated by chemotherapy, resulting in follicle burnout. Following treatment with hAEC exosomes, there is a reduction in the phosphorylation levels of critical proteins, including Akt and FoxO3a, which contributes to the preservation of ovarian reserve

A theory much revisited recently is the role of intrinsic feedback pathways providing endogenous cochlear tissue protection against noise damage

Bue L, Troquier L, Burnouf S, Belarbi K, Van der Jeugd A, Ahmed T, et al

In addition, we demonstrated an enhanced proliferation and self-renewal of G93A-SOD1 mouse epSPCs after in vitro treatment with gold nanovectors loaded with FM19G11, a hypoxia-inducible factor (HIF) modulator 7

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